The background for the article provided for the paper on Wiley.com, explains that cells' autophagy contributes to tumor production while the protein LRPPRC inhibits autophagy and maintains mitochondrial activity. Autophagy is the highly regulated and controlled recycling of wastes within the cell.
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| This image, borrowed from Yale Image Finder (and PubMed) shows the LRPPRC supressor protein (near the center), as well as other proteins, suppressing the expression of genes that trigger autophagy and regulate mitochondria activities. |
The study, headed by Leyuan Liu found that higher levels of LRPPRC had a positive correlation with more severe prostate cancer.
Essentially, elevated levels of the protein prevent the cells from discarding cellular waste, and the resulting imbalance inhibits mitochondrial function.
This does not mean that elevated LRPPRC causes prostate cancer, but it does mean that generally, someone with a lower LRPPRC will survive longer than someone with elevated levels.
Following these findings, drug development will ensue to control levels of LRPPRC in prostate cancer patients to increase the timescale and chances of survival, according to Liu. Additionally, other cancers and their progression may be linked to similar mechanisms.
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